The aim of this overview is to point out MIFs job in several different types of cancers. facts present that MIF is overexpressed in these malignancies in human beings, and contributes for the deregulation in the cell cycle, angiogenesis, and metastasis. Clinical research exhibit that MIF overexpression in these kind of tumors considerably decreases
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We additional showed that CPSI-1306, a little-molecule MIF inhibitor, inhibits The expansion of TNBC cells in vitro. Mechanistic research unveiled that CPSI-1306 induces intrinsic apoptosis by alteration in mitochondrial membrane potential, cytochrome c (Cyt c) release, and activation of various caspases. On top of that, CPSI-1306 inhibits the acti
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